أعرض تسجيلة المادة بشكل مبسط

dc.creator Badimón, Lina
dc.creator Vilahur, Gemma
dc.date 2008-05-09T14:31:17Z
dc.date 2008-05-09T14:31:17Z
dc.date 2007-11-01
dc.date.accessioned 2017-01-31T01:13:40Z
dc.date.available 2017-01-31T01:13:40Z
dc.identifier Cerebrovasc Dis. 24 Suppl 1: 30-39 (2007).
dc.identifier 1015-9770
dc.identifier http://hdl.handle.net/10261/4130
dc.identifier 10.1159/000107377
dc.identifier.uri http://dspace.mediu.edu.my:8181/xmlui/handle/10261/4130
dc.description PMID: 17971637.-- Final full-text version available at: http://dx.doi.org/10.1159/000107377
dc.description Atherosclerosis is a systemic disease that starts early in life, asymptomatically progressing though adulthood, until clinically manifested. In the last few years, experimental, clinical and pathological investigation has led us to a better knowledge of the pathophysiology of atherothrombotic disease. Atherothrombosis is the result of atherosclerosis progression, and its potentially life-threatening clinical consequences include coronary artery disease, cerebrovascular disease and peripheral artery disease. These events are mostly secondary to atherosclerotic plaque disruption and subsequent in situ thrombus formation which may be embolized and dragged by the arterial flow until occluding distal vessels. The demonstrated beneficial role of antiplatelet drugs in reducing the incidence of nonfatal events in many large clinical trials has demonstrated the major role of platelets in the atherothrombotic process in the arterial tree.
dc.description Peer reviewed
dc.format 22195 bytes
dc.format application/pdf
dc.language eng
dc.publisher S. Karger AG
dc.rights closedAccess
dc.subject Atherosclerotic plaque
dc.subject Platelet
dc.subject Atherothrombosis
dc.subject Stroke
dc.title Platelets, Arterial Thrombosis and Cerebral Ischemia
dc.type Artículo


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أعرض تسجيلة المادة بشكل مبسط